By: Neil Z. Miller, medical research journalist and Director of the Thinktwice Global Vaccine Institute
1. What is polio?
Polio is a contagious disease caused by an intestinal virus that may attack nerve cells of the brain and spinal cord. Symptoms include fever, headache, sore throat, and vomiting. Some victims develop neurological complications, including stiffness of the neck and back, weak muscles, pain in the joints, and paralysis of one or more limbs or respiratory muscles. In severe cases it may be fatal, due to respiratory paralysis.
2. How is polio contracted?
Polio can be spread through contact with contaminated feces (for example, by changing an infected baby’s diapers) or through airborne droplets, in food, or in water. The virus enters the body by nose or mouth, then travels to the intestines where it incubates. Next, it enters the bloodstream where “anti-polio” antibodies are produced. In most cases, this stops the progression of the virus and the individual gains permanent immunity against the disease [1].
Many people mistakenly believe that anyone who contracts polio will become paralyzed or die. However, in most infections caused by polio there are few distinctive symptoms [2].In fact, 95 percent of everyone who is exposed to the natural polio virus won’t exhibit any symptoms, even under epidemic conditions [3,4]. About 5 percent of infected people will experience mild symptoms, such as a sore throat, stiff neck, headache, and fever—often diagnosed as a cold or flu [3,5]. Muscular paralysis has been estimated to occur in about one of every 1,000 people who contract the disease [3,6]. This has lead some scientific researchers to conclude that the small percentage of people who do develop paralytic polio may be anatomically susceptible to the disease. The vast remainder of the population may be naturally immune to the polio virus [7].
Injections: Several studies have shown that injections(for antibiotics or other vaccines) increase susceptibility to polio. In fact, researchers have known since the early 1900s that paralytic poliomyelitis often started at the site of an injection [8,9]. When diphtheria and pertussis vaccines were introduced in the 1940s, cases of paralytic poliomyelitis skyrocketed (Figure 1) [10]. This was documented in Lancet and other medical journals [11-13]. In 1949, the Medical Research Council in Great Britain set up a committee to investigate the matter and ultimately concluded that individuals are at increased risk of paralysis for 30 days following injections; injections alter the distribution of paralysis; and it did not matter whether the injections were subcutaneous or intramuscular [14,15].
Figure 1. Polio cases skyrocketed after diphtheria and pertussis vaccines were introduced
Several studies show that injections increase susceptibility to polio. When diphtheria and pertussis vaccines were introduced in the 1940s, cases of paralytic poliomyelitis skyrocketed. This chart shows the average number of polio cases per 100,000 people during five year periods before and after the vaccines were introduced.
Source: National Morbidity Reports taken from U.S. Public Health surveillance reports; Lancet (April 18, 1950), pp. 659-63.
A 1992 study, published in the Journal of Infectious Diseases, validated earlier findings. Children who received DPT (diphtheria, tetanus, and pertussis) injections were significantly more likely than controls to suffer paralytic poliomyelitis within the next 30 days [16]. According to the authors, “this study confirms that injections are an important cause of provocative poliomyelitis [16:444].”
In 1995, the New England Journal of Medicine published a study showing that children who received a single injection within one month after receiving a polio vaccine were 8 times more likely to contract polio than children who received no injections. The risk jumped 27-fold when children received up to nine injections within one month after receiving the polio vaccine. And with ten or more injections, the likelihood of developing polio was 182 times greater than expected [17]. Why injections increase the risk of polio is unclear [18]. Nevertheless, these studies and others [19-24] indicate that “injections must be avoided in countries with endemic poliomyelitis [18].” Health authorities believe that all “unnecessary” injections should be avoided as well [18:1006;24].
Nutritional deficiencies: A poor diet has also been shown to increase susceptibility to polio [25]. In 1948, during the height of the polio epidemics, Dr. Benjamin Sandler, a nutritional expert at the Oteen Veterans’ Hospital, documented a relationship between polio and an excessive use of sugars and starches. He compiled records showing that countries with the highest per capita consumption of sugar, such as the United States, Britain, Australia, Canada, and Sweden (with over 100 pounds per person per year) had the greatest incidence of polio [26]. In contrast, polio was practically unheard of in China (with its sugar use of only 3 pounds per person per year) [26].
Dr. Sandler claimed that sugars and starches lower blood sugar levels causing hypoglycemia, and that phosphoric acid in soft drinks strips the nerves of proper nourishment. Such foods dehydrate the cells and leech calcium from the body. A serious calcium deficiency precedes polio [26-29]. Weakened nerve trunks are then more likely to malfunction and the victim loses the use of one or more limbs [26:146].
Researchers have always known that polio strikes with its greatest intensity during the hot summer months. Dr. Sandler observed that children consume greater amounts of ice cream, soft drinks, and artificially sweetened products in hot weather. In 1949, before the polio season began, he warned the residents of North Carolina, through the newspapers and radio, to decrease their consumption of these products. That summer, North Carolinians reduced their intake of sugar by 90 percent– and polio decreased by the same amount! The North Carolina State Health Department reported 2,498 cases of polio in 1948, and 229 cases in 1949 (data taken from North Carolina State Health Department figures) [26:146;29].
One manufacturer shipped one million less gallons of ice cream during the first week alone following the publication of Dr. Sandler’s anti-polio diet. Soft drink sales were down as well. But the powerful Rockefeller Milk Trust, which sold frozen products to North Carolinians, combined forces with soft drink business leaders and convinced the people that Sandler’s findings were a myth and the polio figures a fluke. By the summer of 1950 sales were back to previous levels and polio cases returned to “normal” [26:146;29].
3. Can polio be treated? Paralytic polio is rarely permanent. Usually there is a full recovery [30-34]. Muscle power begins to return after several days and continues to improve during the next 12-24 months [30-34]. A small percentage of cases will experience residual paralysis. In rare cases, paralysis of the muscles used to breathe can lead to death [5:108;30-34].
Treatment mainly consists of putting the patient to bed and allowing the affected limbs to be completely relaxed. If breathing is affected, a respirator or iron lung can be used. Physical therapy may be required.
4. Does a polio vaccine exist?
In 1947, Jonas Salk, an American physician and microbiologist, became head of the Virus Research Laboratory at the University of Pittsburgh. He was interested in developing a polio vaccine. In 1952, Salk combined three types of polio virus grown in cultures made from monkey kidneys. Using formaldehyde, he was able to “kill” or inactivate the viral matter so that it would trigger an antibody response without causing the disease. That year he began his initial experiments on human subjects. In 1953, his findings were published in the Journal of the American Medical Association. And in April of 1954 the nation’s first polio immunization campaign, directed at school children, was launched [35]. However, shortly thereafter hundreds of people contracted polio from Salk’s vaccine; many died. Apparently, his “killed-virus” vaccine was not completely inactivated [1]. The vaccine was redeveloped, and by August 1955 over 4 million doses were administered in the United States. By 1959, nearly 100 other countries were using Salk’s vaccine [1,35].
In 1957, Albert Sabin, another American physician and microbiologist, developed a live-virus (oral) vaccine against polio. He didn’t think Salk’s killed-virus vaccine would be effective in preventing epidemics. He wanted his vaccine to simulate a real-life infection. This meant using an attenuated or weakened form of the live virus. He experimented with thousands of monkeys and chimpanzees before isolating a rare type of polio virus that would reproduce in the intestinal tract without penetrating the central nervous system. The initial human trials were conducted in foreign countries. In 1958, it was tested in the United States. And in 1963 Sabin’s oral “sugar-cube” vaccine became available for general use [1,35].
5. Which vaccine is in use today?
In 1963, Sabin’s oral vaccine quickly replaced Salk’s injectable shot. It is cheaper to make, easier to take, and appears to provide greater protection, including “herd immunity” in unvaccinated people. However, it cannot be given to people with compromised immune systems [1,35]. Plus, it is capable of causing polio in some recipients of the vaccine, and in individuals with compromised immune systems who come into close contact with recently vaccinated children [1,35-38]. As a result, in January 2000, the CDC “updated” its polio vaccine recommendations, reverting back to policies first implemented during the 1950s: Children should only be given the killed-virus shot. The oral polio vaccine should only be used in “special circumstances [39-41].
6. Are polio vaccines safe? When national immunization campaigns were initiated in the 1950s, the number of reported cases of polio following mass inoculations with the killed-virus vaccine was significantly greater than before mass inoculations, and may have more than doubled in the U.S. as a whole. For example, Vermont reported 15 cases of polio during the one-year report period ending August 30, 1954 (before mass inoculations), compared to 55 cases of polio during the one-year period ending August 30, 1955 (after mass inoculations)– 266% increase. Rhode Island reported 22 cases during the before inoculations period as compared to 122 cases during the after inoculations period — 454% increase. In New Hampshire the figures increased from 38 to 129; in Connecticut they rose from 144 to 276; and in Massachusetts they swelled from 273 to 2027 — whopping 642% increase (Figure 2) [26:140;29:146;42].
Figure 2. Cases of polio increased in the U.S. after mass inoculations
When national immunization campaigns were initiated in the 1950s, the number of reported cases of polio following mass inoculations with the killed-virus vaccine was significantly greater than before mass inoculations, and may have more than doubled in the U.S. as a whole. Source: U.S. Government statistics.
Doctors and scientists on the staff of the National Institutes of Health during the 1950s were well aware that the Salk vaccine was causing polio. Some frankly stated that it was “worthless as a preventive and dangerous to take [26:142].” They refused to vaccinate their own children [26:142]. Health departments banned the inoculations [26:140]. The Idaho State Health Director angrily declared: “I hold the Salk vaccine and its manufacturers responsible” for a polio outbreak that killed several Idahoans and hospitalized dozens more [26:140]. Even Salk himself was quoted as saying: “When you inoculate children with a polio vaccine you don’t sleep well for two or three weeks [26:144;43].” But the National Foundation for Infantile Paralysis, and drug companies with large investments in the vaccine coerced the U.S. Public Health Service into falsely proclaiming the vaccine was safe and effective [26:142-5].
In 1976, Dr. Jonas Salk, creator of the killed-virus vaccine used in the 1950s, testified that the live-virus vaccine (used almost exclusively in the U.S. from the early 1960s to 2000) was the “principal if not sole cause” of all reported polio cases in the U.S. since 1961 [44]. (The virus remains in the throat for one to two weeks and in the feces for up to two months. Thus, vaccine recipients are at risk, and can potentially spread the disease, as long as fecal excretion of the virus continues [45].) In 1992, the Federal Centers for Disease Control and Prevention (CDC) published an admission that the live-virus vaccine had become the dominant cause of polio in the United States [36]. In fact, according to CDC figures, every case of polio in the U.S. since 1979 was caused by the oral polio vaccine [36]. Authorities claim the vaccine was responsible for about eight cases of polio every year [46]. However, an independent study that analyzed the government’s own vaccine database during a recent period of less than five years uncovered 13,641 reports of adverse events following use of the oral polio vaccine. These reports included 6,364 emergency room visits and 540 deaths (Figure 3) [47,48]. Public outrage at these tragedies became the impetus for removing the oral polio vaccine from immunization schedules [36:568;37;38].
Figure 3. Polio vaccine: adverse and serious adverse reactions
In the mid-1990s, during a period of less than five years, there were 13,641 documented adverse reactions to the oral polio vaccine. 6,364 of these were serious enough to require hospital emergency room visits. 540 people died.
Source: Vaccine Adverse Event Reporting System (VAERS); OPV Vaccine Report: Doc. #14
The following story is typical of the damage associated with oral polio vaccines: “Four months ago my son was taken to a local clinic for his polio vaccine. I wasn’t aware that he was going to have one, and would have prevented it if I had known. Unfortunately, he changed from that day — high-pitched screaming, smelly stools, non-stop crying, difficulty in breathing, high temperature, and lethargy. He also lost weight. Weeks of sleepless nights for all of us followed. His development ceased. He had been able to stand and move around, but he went back to remaining in basically whatever position we left him in.
“My wife was six months pregnant at the time, and about a week after our son’s polio vaccine, she began to have headaches, loss of balance, muscular weakness, and frequent tiredness. I panicked because everything seemed to be pointing to polio infection. Then, a week after her continuous headaches began, she had to go to the hospital because there was something wrong with the pregnancy; she lost our daughter.
“I tried to get a polio test, and to find the cause of this tragic series of events, but the medical profession was extremely unhelpful. They laughed at me. I will never know why our son suddenly stopped growing or why his development regressed. I will never know why we lost our daughter. The only thing I am sure about is that the precursor to these events was the polio vaccine.” [From an unsolicited e-mail received by the Thinktwice Global Vaccine Institute—www.thinktwice.com]
Today, fact sheets on polio published by the U.S. Department of Health and Human Services, warn parents that the inactivated polio vaccine (IPV) can cause “serious problems or even death…[49]” The company that manufactures the current inactivated polio vaccine warns that Guillain-Barré Syndrome, a debilitating ailment characterized by muscular incapacitation and nervous system damage, “has been temporally related to administration of another inactivated poliovirus vaccine [3:780].” And although this company makes the claim that “no causal relationship has been established,” it also admits that “deaths have occurred” after vaccination of infants with IPV [3:780]. Yet, like the days of old, despite these “danger alerts,” medical authorities continue to assure parents that the currently available inactivated polio vaccine is both safe and effective.
7. How effective are polio vaccines?
Polio is virtually nonexistent in the United States today. However, according to Dr. Robert Mendelsohn, medical investigator and pediatrician, there is no credible scientific evidence that the vaccine caused polio to disappear [50]. From 1923 to 1953, before the Salk killed-virus vaccine was introduced, the polio death rate in the United States and England had already declined on its own by 47 percent and 55 percent, respectively (Figure 4) [51]. Statistics show a similar decline in other European countries as well [51]. And when the vaccine did become available, many European countries questioned its effectiveness and refused to systematically inoculate their citizens. Yet, polio epidemics also ended in these countries [50].
Figure 4. The polio death rate was decreasing on its own before the vaccine was introduced
From 1923 to 1953, before the Salk killed-virus vaccine was introduced, the polio death rate in the United States and England had already declined on its own by 47 percent and 55 percent, respectively. Source: International Mortality Statistics (1981) by Michael Alderson.
The standards for defining polio were changed when the polio vaccine was introduced. The new definition of a polio epidemic required more cases to be reported. Paralytic polio was redefined as well, making it more difficult to confirm, and therefore tally, cases. Prior to the introduction of the vaccine the patient only had to exhibit paralytic symptoms for 24 hours. Laboratory confirmation and tests to determine residual paralysis were not required. The new definition required the patient to exhibit paralytic symptoms for at least 60 days, and residual paralysis had to be confirmed twice during the course of the disease. Also, after the vaccine was introduced cases of aseptic meningitis (an infectious disease often difficult to distinguish from polio) and coxsackie virus infections were more often reported as separate diseases from polio. But such cases were counted as polio before the vaccine was introduced. The vaccine’s reported effectiveness was therefore skewed (Table 1 and Figure 5) [52,53].
Table 1. Polio or aseptic meningitis?
Cases of polio were more often reported as aseptic meningitis after the vaccine was introduced, skewing efficacy rates. Source: The Los Angeles County Health Index: Morbidity and Mortality, Reportable Diseases.
Figure 5. Polio cases were predetermined to decrease when the medical definition of polio was changed
Source: Congressional Hearings, May 1962; and National Morbidity Reports taken from U.S. Public Health surveillance reports.
The fact that dubious tactics were used to fabricate efficacy rates was corroborated by Dr. Bernard Greenberg, chairman of the Committee on Evaluation and Standards of the American Public Health Association during the 1950s. His expert testimony was used as evidence during Congressional hearings in 1962. He credited the “decline” of polio cases not to the vaccine, but rather to a change in the way doctors were required to report cases: “Prior to 1954 any physician who reported paralytic poliomyelitis was doing his patient a service by way of subsidizing the cost of hospitalization… two examinations at least 24 hours apart was all that was required… In 1955 the criteria were changed… residual paralysis was determined 10 to 20 days after onset of illness and again 50 to 70 days after onset… This change in definition meant that in 1955 we started reporting a new disease… Furthermore, diagnostic procedures have continued to be refined. Coxsackie virus infections and aseptic meningitis have been distinguished from poliomyelitis… Thus, simply by changes in diagnostic criteria, the number of paralytic cases was predetermined to decrease… [52:96,97]”
8. Polio Vaccines and Cancer
In 1959, Bernice Eddy, a brilliant government scientist working in Biologics at the National Institutes of Health, discovered that polio vaccines being administered throughout the world contained an infectious agent capable of causing cancer. When Eddy attempted to report her findings and halt production of these contaminated polio vaccines, her government superiors barred her from publicly revealing the problem. Instead, her lab and equipment were taken away and she was demoted [54,55].
In 1960, Drs. Ben Sweet and M.R. Hilleman, pharmaceutical researchers for the Merck Institute for Therapeutic Research, were credited with discovering this infectious agent – SV-40, a monkey virus that infected nearly all rhesus monkeys, whose kidneys were used to produce polio vaccines. Hilleman and Sweet found SV-40 in all three types of Albert Sabin’s live oral polio vaccine, and noted the possibility that it might cause cancer, “especially when administered to human babies [55,56].” According to Sweet, “It was a frightening discovery because, back then, it was not possible to detect the virus with the testing procedures we had… We had no idea of what this virus would do…” Sweet elaborated: “First, we knew that SV-40 had oncogenic(cancer-causing) properties in hamsters, which was bad news. Secondly, we found out that it hybridized with certain DNA viruses… such that [they]would then have SV-40 genes attached [to them]… When we started growing the vaccines, we just couldn’t get rid of the SV-40 contaminated virus. We tried to neutralize it, but couldn’t… Now, with the theoretical links to HIV and cancer, it just blows my mind [57].”
Further research into SV-40 uncovered even more disturbing information. This cancer-causing virus was not only ingested via Sabin’s contaminated oral sugarcube vaccine, but was directly injected into people’s blood streams as well. Apparently, SV-40 survived the formaldehyde Salk used to kill microbes that defiled his injectable vaccine [58,59]. Experts estimate that between 1954 and 1963, 30 million to 100 million Americans and perhaps another 100 million or more people throughout the world were exposed to SV-40 through ill conceived polio eradication campaigns (Figure 6) [58-60].
Studies published in eminent journals throughout the world appear to confirm that SV-40 is a catalyst for many types of cancer [61-80]. It has been found in brain tumors and leukemia [69-80]. More recently, in 1996, Michele Carbone, a molecular pathologist at Chicago’s Loyola University Medical Center, was able to detect SV-40 in 38 percent of patients with bone cancer and in 58 percent of those with mesothelioma, a deadly type of lung cancer [81-83]. Carbone’s research indicates that SV-40 blocks an important protein that normally protects cells from becoming malignant [83].
Figure 6. Polio Vaccines and simian virus #40
In 1998, a national cancer database was analyzed: 17 percent more bone cancers, 20 percent more brain cancers, and 178 percent more mesotheliomas were found in people who were exposed to SV-40-tainted polio vaccines [84]. The National Institutes of Health created a map showing the geographic distribution of contaminated stock [85]. Using this map, researchers found osteosarcoma bone tumor rates to be 10 times higher than normal in some regions where this tainted vaccine was used (Figure 7) [86,87].
Figure 7. SV-40-tainted polio vaccines: zones of contamination
Between 1954 and 1963, up to 100 million Americans were inoculated with SV-40-contaminated polio vaccines. This chart shows areas of the
country in 1955 where 10 million people received polio vaccines with either no, low, or high amounts of SV-40 in them. Source: National Institutes of Health
Perhaps the most alarming aspect of this ongoing simian virus debacle can be found in other studies suggesting that SV-40, introduced to humans through the polio vaccine, can be passed from human to human and from mother to child. A study of nearly 59,000 women found that children of mothers who received the Salk vaccine between 1959 and 1965 had brain tumors at a rate 13 times greater than mothers who did not receive those polio shots [59:58;88;89].
Another study published in the U.S. medical journal Cancer Research found SV-40 present in 23 percent of blood samples and 45 percent of semen taken from healthy subjects [83:163;90]. Apparently, the virus is being spread sexually and from mother to child in the womb. According to biology and genetics professor Mauro Tognon, one of the study’s authors, this would explain why brain, bone, and lung cancers are on the riseCa 30 percent increase in U.S. brain tumors alone over the past 25 years [83:163;90]—and why SV-40 was detected in brain tumors of children born after 1965 who presumably did not receive polio vaccines containing the virus [83:163;90].
Despite official denials of any correlation between polio vaccines, SV-40, and increased cancer rates [91], by April 2001, 62 papers from 30 laboratories around the world had reported SV-40 in human tissues and tumors [84:10]. The virus was also discovered in pituitary and thyroid tumors, and in patients with kidney disease [84:10,13]. Even the National Cancer Institute issued a statement that SV-40 “may be associated with human cancer [84:11;92].”
Studies yet to be conducted may provide additional clues about the link between contaminated polio vaccines, SV-40, and new diseases. But scientists have their hands full. The latest research has uncovered correlations between polio vaccines, another monkey virus, and AIDS.
8. Polio vaccines and AIDS
SV-40, the cancer-causing monkey virus found in polio vaccines and administered to millions of unsuspecting people throughout the world, was just one of numerous simian viruses known to have contaminated polio vaccines [38:57,58;93;94]. “As monkey kidney culture is host to innumerable simian viruses, the number found varying in relation to the amount of work expended to find them, the problem presented to the manufacturer is considerable, if not insuperable,” one early vaccine researcher wrote to a congressional panel studying the safety of growing live polio-virus vaccine in monkey kidneys [95]. “As our technical methods improve we may find fewer and fewer lots of vaccine which can be called free from simian virus [95].”
According to Harvard Medical School professor Ronald Desrosier, the practice of growing polio vaccines in monkey kidneys is “a ticking time bomb [83:159].” Evidently, some viruses can live inside monkeys without causing harm. But if these viruses were to somehow cross species and enter the human population, new diseases could occur. Desrosier continued: “The danger in using monkey tissue to produce human vaccines is that some viruses produced by monkeys may be transferred to humans in the vaccine, with very bad health consequences [83:159].” Desrosier also warns that testing can only be done for known viruses, and that our knowledge is limited to about “2 percent of existing monkey viruses [83:159].” Craig Engesser, a spokesman for Lederle Laboratories, a large vaccine manufacturing company, acknowledged that “you can’t test for something if you don’t know it’s there [96].”
Virus detection techniques were crude and unreliable during the 1950s, 60s, and 70s when polio vaccines were initially produced and dispensed. It wasn’t until the mid 1980s that new and more sophisticated testing procedures were developed [84:5;96]. That was when researchers discovered that about 50 percent of all African green monkeys—the primate of choice for making polio vaccines—were infected with simian immunodeficiency virus (SIV), a virus closely related to human immunodeficiency virus (HIV), the infectious agent thought to precede AIDS [97-100]. This caused some researchers to wonder whether HIVs may simply be SIVs “residing in and adapting to a human host [101].” It caused others to suspect that SIV may have mutated into HIV once it was introduced into the human population by way of contaminated polio vaccines [59:54+;96-100;102-104].
Vaccine authorities were so concerned about the possibility that SIV was a precursor to HIV, and that polio vaccines were the means of transmission from monkey to human, that The World Health Organization (WHO) convened two meetings of experts in 1985 to explore the data and consider their options [100,105]. After all, SIV was very similar to HIV and occurred naturally in the monkey species predominantly used by vaccine manufacturers [98,100]. Nevertheless, WHO concluded that the vaccines were safe and insisted that vaccination campaigns should continue unabated [100,105].
Shortly thereafter, Japanese researchers conducted their own investigation and found that African green monkeys used to produce polio vaccines had antibodies against SIV [106]. The implication was clear: monkeys used to produce polio vaccines were natural carriers of a virus that looked and acted like HIV, the infectious agent linked to AIDS. In 1989, they recommended that monkeys infected with SIV not be used to make polio vaccines [106].
In 1990, wild chimpanzees in Africa were found to be infected with a strain of SIV that was nearly identical to HIV [107]. Some researchers called it “the missing link” to the origins of human immunodeficiency virus [108]. And since chimpanzees were used to test viruses for potential use in vaccines, and were kept in captivity by research laboratories, they could have been a source of vaccine contamination [109,110]. Scientific concerns were also heightened when researchers found some West Africans who were infected with an SIV-like virus that was a fundamental twin to HIV. They called it HIV-2, and like the initial HIV subtype, it was implicated in the development of AIDS [111]. According to Robert Gallo, an expert on the AIDS virus, some versions of the SIV monkey virus are virtually indistinguishable from some human variants of HIV: “The monkey virus is the human virus.
There are monkey viruses as close to isolates of HIV-2 as HIV-2 isolates are to each other [59:106+].” In May 1991, virus-detection techniques were improved once again, and researchers found SIV DNA in the kidneys of infected monkeys [112]. Minced monkey kidneys were (and still are) used to produce the live polio vaccine [3;59:60]. SIV was also found in the cancer cells of an AIDS victim, and in other people as well [113-115]. To many researchers, this trail of evidence had become too persuasive to deny. Apparently, millions of people were infected with monkey viruses capable of causing AIDS [101], and this cross-species transfer most likely occurred by way of SIV-contaminated polio vaccines [59;84;96-100;102-104;116-119].
10. Didn’t AIDS originate in Africa?
Most historians agree that AIDS originated in Africa [120]. But Salk tested his vaccine in the U.S., and Sabin’s trials were conducted in Eastern Europe and the former Soviet Union [100]. If tainted polio vaccines were responsible for introducing SIV and
HIV into humans, why did the initial cases of AIDS show up on this remote continent?
In March 1951, several years before Drs. Jonas Salk and Albert Sabin would scuffle over whose vaccine was the true prophylactic, Dr. Hilary Koprowski announced at a medical conference that he had become the first doctor in history to test a polio vaccine on humans. His “volunteers” included several institutionalized children with mental handicaps. They drank the vaccine in chocolate milk [121].
From 1957 to 1960, after years of tinkering with monkey kidneys and polio germs, Koprowski tested his own experimental polio vaccine on 325,000 equatorial Africans, including 75,000 citizens of Leopoldville, Belgian Congo (now Kinshasa, Zaire)
[59:59;121]. Called by drums, rural natives traveled to local villages where they had a liquid vaccine squirted into their mouths [122]. Ninety-eight percent of the vaccine recipients were infants and toddlers [121]. The youngest children received 15 times the adult dosage [103:98]. Though Koprowski claimed he had the backing of the World Health Organization, WHO denied sanctioning the large-scale trials [123].
In 1959, Dr. Albert Sabin reported in the British Medical Journal that Koprowski’s polio vaccine used in the African trials contained an “unidentified” cell-killing virus [124]. It was never identified. However, in 1986 the earliest known blood sample containing antibodies against HIV was traced back to 1959. The serum came from a patient visiting a clinic in Leopoldville [125]. There is no evidence that HIV infected humans before 1959 [126,127]. Gerald Myers, a genetic sequencing expert with Los Alamos National Laboratories in New Mexico, tracked the evolution of HIV and confirmed that today’s major subtypes of the AIDS virus in humans appear to have arisen as recently as 1960 [128].
Koprowski’s vaccine was not approved for human use, so it was discontinued in 1960 following the African trials [100]. Thus, it was only administered to inhabitants of the Belgian Congo, Rwanda and Burundi [104,121]Cthe precise area where high levels of HIV infection were identified by researchers 30 years later [129]. Furthermore, the AIDS virus is known to infect mucous cells, prevalent in the mouth [59:60]. The African vaccines were squirted into people’s mouths. Could squirting an HIV contaminated polio vaccine into people’s mouths cause AIDS? According to Tom Folks, chief retrovirologist at the CDC, “Any time a person has a lesion in his mouth, then there could be transmission” of the virus [59:60]. Dr. Robert Bohannon of Baylor College of Medicine maintains that the process of squirting the polio vaccine into people’s mouths would tend to aerosolize some of the liquid. Tiny drops could then go directly into the lungs, and from there to the blood cells susceptible to infection [59:60]. This would have been an efficient mode of HIV transmission [100].
Disease experts believe that the average time between HIV infection and the development of AIDS is 8-10 years [100]. If the African polio vaccine was indeed contaminated with SIV/HIV, initial outbreaks of AIDS would have occurred from the mid-
1960s to early 1970s. This period accurately coincides with the emergence of AIDS in equatorial Africa [130].
11. Test the polio vaccines
Authorities are reluctant to acknowledge the possibility that medical scientists, preoccupied with growing polio vaccines in virus-laden monkey kidneys, may have been responsible for bringing about the AIDS pandemic. For example, Dr. David Heymann, who heads the World Health Organization’s Global Program on AIDS, flatly stated that “the origin of the AIDS virus is of no importance to science today [59:106+].” William Haseltine, a Harvard pathology professor and AIDS researcher also believes that any discussion about the origin of AIDS is distracting and nonproductive. “It’s not relevant,” and “I’m not interested in discussing it [59:106+].” Jonas Salk won’t discuss the subject either. He is now working on an AIDS vaccine [59:55]. Albert Sabin believes “you can’t hang Koprowski with that [59:60].” And Koprowski dismissed the idea with a laugh, then later claimed “this is a highly theoretical situation [59:106+].” However, samples of the polio vaccines used in Africa are kept in freezers at the Wistar Institute where Koprowski did much of his research. They could be tested [59:106+].
Tom Folks of the CDC thinks it’s a good idea to test the seed stocks of polio because “any time we can learn more about the natural history [of AIDS], it helps us understand the pathogenesis and…the transmission [59:106+].” Robert Gallo also thinks it’s
important to determine whether a monkey virus sparked AIDS. Questions like this “are of more than academic interest because answering them may help avoid future zoonotic catastrophes—that is, transmission of disease from lower animals to humans [131].” Responding to these concerns, some AIDS researchers formally requested samples of the original polio vaccine seed stocks. But the government will neither release nor test them because there are “only a small number of vials” of the material, and tests “might use it all up [59:108].”
12. AIDS within the gay community
If AIDS originated in Africa via contaminated polio vaccines, how did this disease spread to male homosexuals in America? In 1974, clinics in New York and California began experimental treatments for gay men afflicted with herpes. Therapy consisted of multiple doses of the live polio vaccine [132]. As noted earlier, this vaccine was produced in the kidneys of the African Green monkey, a known reservoir for simian immunodeficiency virus (SIV), a likely precursor to HIV [59;84;97-104]. Beginning in the early 1980s, simultaneous outbreaks of Kaposi sarcoma and serious opportunistic infections (later associated with AIDS) were reported among homosexual men, especially in New York City, San Francisco, and Los Angeles [99]. This time span coincides with the average incubation period between HIV infection and the development of AIDS [100].
In 1982, the CDC concluded that such outbreaks “strongly suggests the occurrence of a single epidemic of underlying immunosuppression… [133]” The following year, HIV was identified as the causative agent [99]. And in 1992, Lancet published the first
scientific explanation showing how repeated doses of SIV contaminated polio vaccines may have seeded HIV among American homosexual men [99].
13. AIDS with no identified risk factor (NIR)
Another unusual event occurred in the 1980s. Hundreds of people diagnosed with AIDS had no identified risk factor (NIR) [134]. They did not engage in risky behaviors related to AIDS infection. The CDC also listed numerous children as NIR [134]. Some parents believe HIV-contaminated polio vaccines infected their loved ones [135].
On February 12, 1994, Bruce Williams filed a civil suit against the American Cyanamid Company, claiming its polio vaccine caused his daughter’s illness. The suit alleges that “the live oral poliovirus vaccine was produced, tested, and approved by the United States Food and Drug Administration pursuant to measures inconsistent with accepted standards of medical practice.” The lawsuit also asserts that “the product was FDA approved despite the known presence of contaminants, including retroviruses such as HIV [136].”
Walter Kyle, the Williams’ lawyer, identified the specific lots of vaccine the child received, but the CDC and federal health officials have refused to test them [134:106]. Kyle believes “The CDC could disprove my entire hypothesis by testing the vaccines they have in their possession. The fact that they haven’t done so is evidence there’s something wrong with the vaccine [134:106].”
Some researchers believe the true number of NIR cases could be in the thousands [134,137]. When health officials examine people with AIDS, they try to identify a risk factor. If a patient admits he once had unprotected sex, that becomes his factor, even though there’s no proof that is how he was infected [134].
The evidence implicating polio vaccines grown in monkey kidneys with our current epidemics of cancer and AIDS continues to grow. But what if polio vaccines were produced in cow serum? Would that make a difference?
~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~
For complete list of this articles sources click on this text
~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~
The above is a 3 part article (combined here) that was initially published on VaxTruth. TLB highly recommends you visit this website for more great/pertinent articles and information concerning the dangers of vaccines.
See featured article and read comments here: http://vaxtruth.org/2012/03/the-polio-vaccine-part-1-2/
OK